Overview

The Beta Cell Responsiveness to Glucose-dependent Insulinotropic Polypeptide (GIP) With and Without Sulfonylurea in Patients With Type 2 Diabetes

Status:
Completed

Trial end date:
2007-04-01

Target enrollment:

Participant gender:

Summary

The investigators hypothesize that the impaired insulinotropic effect of the incretin hormone GIP may be due to inadequate sensitization and ATP induced closure of beta cell K-ATP channels. By closing the channels through the use of sulfonylurea (SU) we hope to restore the insulinotropic effect of GIP.

Phase:

Phase 2

Details

Lead Sponsor:

University Hospital, Gentofte, Copenhagen

Treatments:

Gastric Inhibitory Polypeptide